Ptosis vs Hooded Eyes: How to Tell Them Apart (and Fix It)

Educational content reviewed against the published dermatology and oculoplastic surgery literature. Citations link to peer-reviewed sources on PubMed, PMC, and major medical journals. This article is informational and is not a substitute for a consultation with a qualified ophthalmologist or oculoplastic surgeon.

Your eyelids feel heavier than they used to. Eyeshadow disappears the moment you open your eyes. People keep asking if you’re tired even when you’ve slept nine hours. So you start searching — and you run into two words that look like they mean the same thing: hooded eyes and ptosis.

They don’t. And the difference matters more than almost anyone tells you, because one is a cosmetic feature you can address at home, and the other is a medical condition that sometimes signals something serious. Mixing them up means either spending money on the wrong solution — or, worse, ignoring a symptom that needs a doctor.

This guide walks you through exactly how to tell which one you have, what the published clinical literature says about the causes of each, and what actually works (and what doesn’t) for both — with every claim linked to a peer-reviewed source.

Hooded Eyes vs Ptosis: The Quick-Look Comparison

That table is the headline answer. The rest of this article explains why the distinction works clinically, gives you a 30-second test you can do in front of a mirror, and walks through what the dermatology and oculoplastic surgery literature actually says works for each.

What Are Hooded Eyes, Exactly?

Hooded eyes happen when the skin above your eyelid — the area between the brow bone and the lash line — carries enough excess tissue or laxity that it folds down and partially covers the natural crease. The medical term for the age-related version is dermatochalasis, defined in the oculoplastic literature as redundant, lax upper-eyelid skin caused by progressive loss of dermal collagen and elastin (EyeWiki: Blepharoptosis & Dermatochalasis).

The key word is skin. The eyelid muscle underneath is working perfectly. The eye itself opens to its full normal width. It’s the layer above the lid that’s drooping — which is why the right intervention targets the dermis, not the muscle.

What causes hooded eyes (the published mechanisms)

Hooded eyes are not one problem — they’re the visible result of several biological changes converging on the thinnest skin on your body. The eyelid skin is roughly 0.3–0.5 mm thick versus 1.5–2 mm on the cheek, which is why it shows aging first.

• Genetics. Some people are simply born with a low brow bone or extra upper-lid skin. If your parents had hooded eyes at 30, you probably will too.
• Collagen and elastin loss. Dermal collagen production declines roughly 1% per year after age 20, accelerating dramatically around perimenopause. Loss of dermal extracellular matrix proteins (collagen, elastin, glycosaminoglycans) is the primary driver of skin laxity (Borkow, Curr Chem Biol, 2014).
• Mitochondrial decline. Aged dermal fibroblasts show reduced mitochondrial number, lower oxidative phosphorylation rates, and decreased NAD+ pools — meaning the cells responsible for building collagen have less cellular energy to do the job (Oblong et al., Aging Cell, 2020).
• UV damage. Cumulative sun exposure breaks down collagen fibres and accelerates laxity, especially in people who skip SPF on the eyelids.
• Sleep, rubbing, and lifestyle. Side- and stomach-sleeping, vigorous eye-rubbing (allergies are a big one), and dehydration all stretch the delicate skin over time.
• Hormonal shifts. Menopause, in particular, accelerates collagen loss and can make hooding noticeable within a year or two.

What none of these involve is the muscle that lifts your eyelid. That muscle is fine. The skin above it is the problem — which is why most cases of hooded eyes can be addressed at home, without surgery, if you target the right thing in the right layer.

What Is Ptosis?

Ptosis (pronounced “toe-sis” — the “p” is silent) is when the upper eyelid itself sits lower than it should. The eyelid margin — the edge where your lashes are — falls over part of the iris or even the pupil.

This is not a cosmetic issue. It’s a mechanical or neurological problem with the apparatus that opens your eye. Two muscles do that work: the levator palpebrae superioris (the main one, innervated by the third cranial nerve) and a smaller sympathetically innervated muscle called Müller’s muscle. When either of them, or the nerve that controls them, fails — the lid drops (American Academy of Ophthalmology: Acquired Ptosis).

The five clinical types of ptosis

• Aponeurotic ptosis — the most common type by a wide margin. A retrospective series found aponeurotic ptosis accounted for 73% of adult ptosis cases (PMC9834322, 2022). The tendon of the levator muscle stretches or detaches with age, eye-rubbing, contact lens wear, or after eye surgery.
• Myogenic ptosis — the muscle itself is weak. Conditions like myasthenia gravis (an autoimmune disorder) and certain muscular dystrophies cause this. The same series above identified myasthenia gravis in 17% of ptosis patients (PMC9834322). Signature clue: the droop gets worse as the day goes on or with fatigue.
• Neurogenic ptosis — a nerve problem. Causes include third cranial nerve palsy and Horner’s syndrome, which can come from issues as serious as a carotid artery dissection or a tumour pressing on the sympathetic nerve chain (StatPearls: Ptosis Correction).
• Mechanical ptosis — something is physically weighing the lid down. A cyst, a tumour, severe swelling, or scar tissue.
• Congenital ptosis — present from birth. The levator muscle didn’t develop properly.

This is why a sudden droop, a droop in only one eye, or a droop that comes with double vision, headache, or a different-sized pupil is a medical situation, not a cosmetic one. We’ll come back to those red flags below.

The 30-Second Self-Test: Hooded Eyes or Ptosis?

Stand in front of a well-lit mirror and look straight ahead with a relaxed face. Don’t lift your brows.

1. Find the edge of your upper eyelid — the line where your lashes grow.
2. Look at where that edge sits relative to your iris (the coloured part of your eye). Clinicians measure this as the marginal reflex distance (MRD1) — in a healthy adult it’s typically 4–5 mm from the pupil to the lid edge (Ptosis — clinical assessment).
3. Now compare it to where the “heavy” feeling is.

If the lid margin (with the lashes) is in its normal position, but the skin above it is folding down and covering the crease — that’s hooded eyes. Lift the brow bone gently with one finger; the heaviness lifts away because the lid itself was already in the right place.

If the lid margin itself is sitting low — covering more than the top third of the iris, or even creeping toward the pupil — that’s ptosis. Lifting your brow won’t fix it; the lid edge is still low.

An even quicker check: look at a recent photo of yourself versus a photo from 5–10 years ago. If both eyelids have changed gradually and symmetrically and the lid margin still sits in roughly the same place, you’re almost certainly looking at hooding. If one eye looks markedly different from the other, or the lid edge has clearly moved down, you’re looking at probable ptosis.

When You Need to See a Doctor — Today, Not Eventually

Skip every at-home solution and book an ophthalmologist (or go to A&E) if any of the following apply:

• Sudden onset — the droop appeared in hours or days, not years.
• Asymmetry — one eyelid is clearly lower than the other.
• Double vision, blurred vision, or a change in your visual field.
• A different-sized pupil on the affected side — a classic sign of Horner’s syndrome or third nerve palsy.
• Droop that gets worse as the day goes on or after exertion — potentially myasthenia gravis.
• Headache, neck pain, or facial numbness alongside the droop.
• Difficulty swallowing or generalised muscle weakness.

If none of those apply, and what you’re looking at is gradual, symmetrical heaviness of the upper-eye area — you’re almost certainly dealing with cosmetic hooding, and you have real, science-backed options that don’t involve a scalpel.

Why Surgery Is Rarely the Right Answer for Hooded Eyes

Upper-lid blepharoplasty — the surgical removal of excess upper-lid skin — is the most aggressive intervention for hooded eyes. Surgeons will often present it as a clean, predictable, one-and-done fix. The published complications literature tells a more complicated story.

The complications nobody mentions on the consultation page

A 10-year retrospective review of 892 blepharoplasty cases by a single surgeon found that 26.5% of patients developed dry eye symptoms or chemosis after surgery, with risk factors including concurrent upper and lower lid surgery, postoperative lagophthalmos (incomplete eyelid closure), and skin-muscle flap technique (Prischmann et al., JAMA Facial Plast Surg, 2013).

The published list of recognised blepharoplasty complications in the peer-reviewed literature includes (Pacella & Codner, Semin Plast Surg, 2010; Oestreicher & Mehta, Plast Surg Int, 2012):

• Dry eye disease — from disrupted tear film mechanics, reduced blink rate, and damage to the orbicularis oculi muscle (Hu et al., Int J Ophthalmol, 2020).
• Lagophthalmos — the inability to fully close the eyelid, leading to chronic exposure keratitis.
• Eyelid ptosis as a complication — surgery to fix hooding can cause ptosis if the levator aponeurosis is inadvertently disrupted.
• Ectropion and lid retraction — the lid pulled away from the eye, creating a permanent “wide-eyed” appearance and exposing the eye to drying.
• Asymmetric eyelid creases — one fold higher than the other, irreversible without revision surgery.
• Hematoma — in the worst form (post-septal/retrobulbar), this can cause optic nerve compression and permanent vision loss. Rare but documented.
• Diplopia — double vision from disruption of extraocular muscles.
• Scarring, eyelid furrow deformities, and canthal webbing.
• Permanent vision loss — the most feared complication. The literature reports an incidence of orbital hemorrhage with permanent visual loss at approximately 0.0045%. Low — but not zero.

The other costs surgeons don’t put on the price list

• $3,000–$7,000+ in the United States for upper blepharoplasty alone, almost always out of pocket since insurance only covers it when there’s documented visual field obstruction.
• 1–2 weeks of visible bruising and swelling, with full healing taking up to 6 months.
• Permanent and irreversible — once skin is excised, there’s no putting it back. Over-resection is a known cause of dry eye and lagophthalmos that can’t be undone.
• The procedure doesn’t stop aging. The remaining skin continues to lose collagen and elastin. Most patients report visible re-laxity within 7–10 years.

Tear-trough fillers and Botox brow lifts are the other commonly suggested “less invasive” options. They have their own published complication profiles — filler migration, the Tyndall effect (a persistent blue-grey discoloration), vascular occlusion in rare cases, and dependency cycles ($600–$1,400 every 6–12 months for the rest of your life).

For mild to moderate hooding — which is most hooding — the question isn’t “surgery or nothing.” It’s “which non-surgical approach actually works on the right layer of the problem.”

What Actually Works: Targeting the Dermis Where Collagen Is Built

Hooded eyes are a problem of dermal structural decline. To meaningfully change the appearance of hooded eyes without surgery, an active ingredient has to do two things:

1. Reach the dermis — the layer where collagen and elastin are produced. The stratum corneum (the outermost layer of skin) blocks more than 95% of topical molecules, especially the larger peptides and DNA-derived actives that work best for skin remodeling (Microneedling and transdermal delivery, Pharmaceutics, 2022).
2. Trigger collagen synthesis once it’s there — either by signaling fibroblasts directly or by providing the cellular energy fibroblasts need to do their job.

This is the core scientific case for micro-infusion as a delivery method, and for the specific actives that follow. Each link below points to a peer-reviewed study.

GHK-Cu (Copper Tripeptide-1): the most-studied skin-remodeling peptide

GHK-Cu is a copper-binding tripeptide naturally present in human plasma that declines with age. Forty years of research support its role in skin regeneration, collagen synthesis, and wound healing.

• In a placebo-controlled trial of 71 women with photoaged skin, twelve weeks of GHK-Cu facial cream increased skin density and thickness, reduced laxity, improved clarity, and reduced fine line and wrinkle depth (Pickart et al., BioMed Research International, 2015).
• In a separate 12-week study of 41 women with mild to advanced photodamage around the eyes, a GHK-Cu eye cream outperformed both placebo and a vitamin K cream, reducing lines and wrinkles, improving overall appearance, and increasing skin density and thickness (Pickart et al., 2015).
• Compared head-to-head against vitamin C cream and retinoic acid in a thigh-skin biopsy trial, GHK-Cu produced collagen increases in 70% of women treated, versus 50% for vitamin C and 40% for retinoic acid (Pickart & Margolina, Int J Mol Sci, 2018).
• Mechanistically, GHK-Cu modulates over 4,000 human genes, essentially shifting fibroblast gene expression toward a younger profile. It increases collagen, dermatan sulfate, chondroitin sulfate, and decorin synthesis, and modulates the activity of matrix metalloproteinases that break down dermal collagen (Pickart & Margolina, 2018).

PDRN (Polydeoxyribonucleotide): regeneration via the salvage pathway

PDRN is a low-molecular-weight DNA-derived biopolymer (typically extracted from salmon trout testes) that activates the adenosine A2A receptor on skin cells, triggering tissue regeneration and an anti-inflammatory cascade.

• A 2025 systematic review covering decades of clinical data confirmed that PDRN/PN formulations promote skin regeneration, reduce inflammation, improve skin texture, prevent scar formation, and mitigate wrinkles — with an excellent safety profile (Marques et al., Biomolecules, 2025).
• Mechanistically, PDRN supplies the building blocks (purines and pyrimidines) that cells need to repair their own DNA via the salvage pathway, while simultaneously activating A2A receptor signaling that drives fibroblast proliferation and collagen production (PN vs PDRN comparative review, Pharmaceuticals, 2025).
• An active clinical trial at the University of Taubaté is currently testing PDRN delivered specifically by microneedling to periocular wrinkles — the same skin region affected by hooded eyes (NCT07280637, ClinicalTrials.gov).

NAD+ (Nicotinamide Adenine Dinucleotide): the cellular energy currency

NAD+ is the central cofactor for ATP synthesis, DNA repair, and sirtuin activation in every cell in your body — including the dermal fibroblasts that build collagen. Skin NAD+ levels decline by approximately 50% between age 20 and age 50, contributing to the metabolic dysfunction that makes aged skin slower to repair itself (Oblong et al., Aging Cell, 2020).

• A 2024 study in Antioxidants demonstrated that exogenous NAD+ enhances fibroblast wound healing, increases Ki67 expression (a marker of cell proliferation), and activates sirtuins and autophagy — the cellular processes that drive tissue repair (Yoon et al., Antioxidants, 2024).
• NAD+ precursors restore mitochondrial number, membrane potential, and oxidative phosphorylation in fibroblasts from older donors, reversing the metabolic deficit that underlies aged-skin behavior (Oblong et al., 2020).
• A 2025 study in human skin fibroblasts showed that boosting NAD+ levels suppresses cellular senescence, supports extracellular matrix integrity, and accelerates wound healing under oxidative stress conditions that mimic photoaging (Antioxidants, 2025).

The clinical logic is straightforward: peptides like GHK-Cu provide the genetic instructions for skin renewal. NAD+ provides the cellular energy to execute them. One without the other is half the equation.

Why micro-infusion at 0.25mm is the right delivery system for hooded eyes

The single biggest reason most peptide eye creams disappoint is that they never reach the dermis. The peer-reviewed transdermal delivery literature is consistent on this: large, charged molecules like peptides and PDRN have very limited skin penetration when applied topically (PDRN topical delivery review, 2025).

Micro-infusion solves the delivery problem mechanically. Tiny needles — in the case of at-home eye-area systems, calibrated to 0.25mm — create temporary microchannels in the skin that bypass the stratum corneum. This both stimulates the skin’s natural wound-healing cascade (which itself induces collagen production) and allows applied actives to reach the dermal layer where fibroblasts live (Microneedling: Collagen Induction Therapy review, J Dermatol Dermatologic Surg, 2021).

The clinical literature is specific about depth: 0.25–0.5mm is the established at-home safe range for fine lines and early wrinkles. Deeper needles (0.5–2.5mm) are reserved for clinical settings because they carry risk of bleeding, infection, scarring, and post-inflammatory hyperpigmentation. For the delicate skin of the eye area — the thinnest skin on the body — 0.25mm is the sweet spot: deep enough to reach the upper dermis where collagen remodeling signals are most active, shallow enough to avoid bleeding, bruising, and downtime.

Microneedling alone has been shown in clinical research to increase collagen production by up to 400% over a treatment course, with measurable wrinkle-depth reduction visible after 3–6 months of consistent treatment. When the same micro-channels also deliver active peptides and DNA-derived regenerative molecules, the effects compound — you’re both stimulating the wound-healing cascade and handing the fibroblasts the exact signals and energy they need to remodel the dermis.

The Botanique Paris Approach to Hooded Eyes

This is exactly why we built the Micro-Infusion System for Upper Eyes: to deliver the three most-researched skin-remodeling actives — GHK-Cu, PDRN, and NAD+ — into the dermal layer where hooded eyes actually form, using precision 0.25mm needles calibrated specifically for the delicate upper-eye area.

The mechanism is straightforward and matches everything the published literature says works:

• The 0.25mm micro-channels bypass the stratum corneum that blocks ~95% of topical actives, while triggering the skin’s own collagen-induction wound-healing response.
• GHK-Cu reaches the dermis at concentrations that signal fibroblasts to remodel the extracellular matrix, increase collagen and elastin production, and shift gene expression toward a younger profile.
• PDRN activates A2A receptor signaling and supplies nucleotides for tissue regeneration in the upper-eye skin where it’s applied.
• NAD+ restores cellular energy in dermal fibroblasts that have lost up to half their baseline NAD+ pool with age — powering the collagen synthesis the peptides are signaling for.

The system is currently endorsed by 816 unpaid clinicians — a number that’s climbed steadily as more practitioners encounter it — and is backed by our 365-day money-back guarantee. Five minutes, twice a week, at home. No bruising. No downtime. No scalpel. No skin you can never get back.

Most consistent users report visible firming and lifting of the upper-eye area within 4–8 weeks, with continued improvement over the following months as collagen remodeling compounds. Compared to the $3,000–$7,000 cost, 1–2 weeks of visible bruising, and permanent risks of upper-lid blepharoplasty — or the ongoing $600–$1,400 hamster-wheel of fillers and Botox — it’s a different category of intervention entirely. The skin itself changes. There’s nothing to dissolve, nothing to redo, nothing migrating, and nothing being permanently removed from a part of your face that you can never put back.

See the Micro-Infusion System for Upper Eyes →

What Doesn’t Work for Hooded Eyes (Despite the Marketing)

• Eye creams alone. A peptide cream that can’t penetrate the stratum corneum is, biochemically, a $90 moisturiser. The peptide molecule is too large and too charged to cross the skin barrier in meaningful concentration without a delivery method.
• Cooling rollers and jade gua sha. They feel nice and reduce temporary puffiness through lymphatic drainage. They don’t address skin laxity or stimulate dermal remodeling.
• Eye-lift exercises. Hooded eyes are a skin issue, not a muscle weakness issue. Exercising muscles that are already working won’t lift skin sitting on top of them. (This is also true of facial yoga claims more broadly.)
• Tape and adhesive strips. Hide the issue for an evening. Don’t change anything underneath, and repeated tugging can accelerate skin laxity.
• Generic at-home derma rollers. Without a calibrated, clinically-formulated serum delivered through the microchannels, the roller alone provides only a fraction of the benefit. Worse, unregulated rollers above 0.5mm carry real risks of infection, scarring, and pigmentation changes.

How to Address Ptosis (If That’s What You Have)

If your self-test pointed to ptosis — the lid margin itself sitting low — the path looks completely different. We’re going to be honest about this: at-home cosmetic devices, including ours, will not fix true ptosis. The muscle or nerve issue is below the skin entirely. Treating it with skincare while the underlying cause goes undiagnosed is the wrong move.

Step one is always a clinical evaluation. An ophthalmologist or oculoplastic surgeon will measure the marginal reflex distance, assess your levator function, and rule out the neurological causes that need urgent attention.

Treatment depends entirely on the cause:

• Aponeurotic ptosis is generally repaired with a procedure that re-tightens or re-attaches the stretched levator tendon. Insurance often covers this when the droop interferes with the visual field.
• Myogenic ptosis from myasthenia gravis is treated medically — the droop often improves dramatically with the right medication.
• Neurogenic causes require treating the underlying nerve problem, which can range from controlling diabetes to neurosurgical intervention depending on the source.
• Recently approved prescription eye drops (oxymetazoline 0.1%, FDA-approved 2020) temporarily stimulate Müller’s muscle and can lift the lid 1–2 mm for several hours in mild aponeurotic ptosis.

Can You Have Both at the Same Time?

Yes — and this is more common than people realise after age 50. The skin above the lid loosens (hooding) at the same time the levator tendon stretches (mild aponeurotic ptosis). When this happens, an oculoplastic surgeon will often recommend addressing both at once: a combined upper-lid blepharoplasty plus ptosis repair.

If yours is mild on both counts, a non-surgical strategy can still help with the hooded component while you decide whether to address the ptosis. A medically-supervised approach to the ptosis combined with a science-backed at-home approach to the hooding (micro-infusion of GHK-Cu, PDRN, and NAD+) is, for many people, the optimal middle path — addressing each problem with the right intervention rather than asking surgery to do everything.

Frequently Asked Questions

How can I tell if my eyelids are hooded or drooping?

Look at where the edge of your upper eyelid (the lash line) sits relative to your iris. If the lid edge is in its normal position but the skin above it folds down over the crease, that’s hooded eyes. If the lid edge itself is low — covering more than the top third of the iris — that’s ptosis. Clinicians use a measurement called the marginal reflex distance (MRD1) to formalise this; in healthy adults it’s typically 4–5 mm.

Is ptosis dangerous?

Most ptosis is benign age-related stretching of the levator tendon — about 73% of cases per published series. However, ptosis can also be the first sign of conditions like myasthenia gravis (around 17% in one series), Horner’s syndrome, or third cranial nerve palsy. A sudden droop, a droop in only one eye, or a droop accompanied by double vision, headache, or a different-sized pupil should be evaluated urgently by a doctor.

Can hooded eyes be fixed without surgery?

Mild to moderate hooded eyes respond well to non-surgical approaches that target collagen and skin laxity in the upper-lid area — particularly micro-infusion (which delivers peptides through the skin barrier), microcurrent, and consistent red light therapy. The dermatology and oculoplastic literature is consistent that surgery is the most aggressive option, not the only one.

Will eye creams alone fix hooded eyes?

Almost certainly not. The eyelid skin’s stratum corneum blocks more than 95% of topically-applied peptides and DNA-derived actives from reaching the dermis where collagen is built. Active ingredients need a delivery method — micro-infusion or microcurrent — to reach the layer that actually changes how the skin behaves.

How long does it take to see results from non-surgical hooded-eye treatment?

Most consistent users of micro-infusion systems report visible firming of the upper-eye area within 4 to 8 weeks of regular use. Collagen remodeling is a months-long biological process, so the most dramatic improvements typically appear at the 12–16 week mark and continue to compound with consistent use.

What are the risks of upper-lid blepharoplasty?

Published complications include dry eye disease (around 26% in a 10-year retrospective series of 892 cases), lagophthalmos (incomplete eyelid closure), eyelid ptosis as a complication of surgery, ectropion, asymmetric creases, hematoma, diplopia, scarring, and in rare cases permanent vision loss from orbital hemorrhage. The procedure is also irreversible — once skin is removed, it cannot be replaced — and most patients see re-laxity within 7–10 years.

What’s the difference between hooded eyes and dermatochalasis?

They’re essentially the same thing — dermatochalasis is the medical term for excess upper-lid skin caused by progressive loss of dermal collagen and elastin. “Hooded eyes” is the everyday term, often used to describe both the genetic version (present from young adulthood) and the age-related version (which technically is dermatochalasis).

Why does the Micro-Infusion System use 0.25mm needles specifically?

0.25mm is the established at-home safe depth for the delicate eye-area skin in the dermatology literature. It reaches the upper dermis where collagen remodeling signals are most active without causing bleeding, bruising, or downtime. Deeper needles (0.5mm+) are reserved for clinical settings due to higher risk of scarring, infection, and post-inflammatory hyperpigmentation. For the eye area, going deeper is not better — it’s riskier.

The Bottom Line

Hooded eyes and ptosis can look almost identical in a mirror, but they are not the same problem and they don’t respond to the same solutions.

If the skin above your eyelid is what’s heavy — gradual, symmetrical, no vision changes — you’re looking at hooded eyes, and the published clinical literature points to a clear strategy: deliver collagen-stimulating actives (GHK-Cu, PDRN, NAD+) into the dermis where fibroblasts can actually use them, via a calibrated 0.25mm micro-infusion system. That’s exactly what the Botanique Paris Micro-Infusion System for Upper Eyes was built to do — without the cost, irreversibility, or documented complication profile of upper-lid blepharoplasty.

If the eyelid itself is low — sudden, asymmetrical, or paired with any visual or neurological symptom — please see an ophthalmologist before you spend a cent on cosmetic devices. Most ptosis is benign, but it’s the kind of “most” you want a doctor to confirm.

Either way, you now know what you’re actually dealing with — which is the part nobody tells you when you start searching at midnight wondering why your eyes look so heavy.

Try the Micro-Infusion System for Upper Eyes →

365-day money-back guarantee · Endorsed by 816+ clinicians · 0.25mm precision delivery

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This article is for general educational purposes and does not constitute medical advice. If you suspect you have ptosis, please consult an ophthalmologist or oculoplastic surgeon for diagnosis and treatment options. The Botanique Paris Micro-Infusion System is intended for cosmetic use on the skin and is not designed to diagnose, treat, cure, or prevent any medical condition. Individual results vary.